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anesthesia monitoring

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Normal VT
10-20 mL/kg in most domestic species
Alveolar Ventilation ( VA) =
RR x (VT– VD)
Hypercapnia: PaCO2 > 45 mmHg
§ From 45 - ˜100 mmHg it is a respiratory stimulant, § CNS stimulant and vasodilator § In excess of 100mmHg it is an anesthetic & CNS depressant
PAO2 =
[(Patm-PH20) x FiO2] - PaCO2 /0.8
“Cyanosis” requires >
5gm/dL of deoxygenated Hb
CaO2=
(Hg x %Sat x 1.34)+ (0.003 x PaO2)
Apnea Monitor
detects air flow, rate only, alarms w/apnea §continuous, auditory signal
Ventilometry
•Measures the volume of a breath (tidal volume) §simple turbine-propelled vWright’s respirometer §computerized, flow-generated
Pulse oximetry
•Detects infrared light absorption at two wavelengths, and calculates saturation of hemoglobin with O2 •Contains algorithm to subtract non-pulsatile reflectance •PaO2 = SpO2 – 30 (estimate)
MAP needed for
1.brain
2.renal
3.muscles
Cerebral and coronary (50-60 mmHg) §Renal and mesenteric (60-70 mmHg) §Muscle compartments (>70 mmHg)
pulse Not palpable lower than MAP of about
40
sphyngomanometry:
uses a cuff to occlude arterial supply to a known pressure, and define the return of flow (systolic)
oscilometric:
uses cuff, but computer detects oscillations of artery. (S, D and M)
calculated MAP=
D+1/3(S-D)
Pulse Oximetry⬢Accuracy
⬢Fair SpO2 ,<> 75-90% ⬢Overestimation SpO2< 70% ⬢ Underestimation SpO2 > 90%
Heart Rate limits on CO
⬢Heart rate directly contributes to CO up to a point, then it decreases CO: §Decreasing preload (most of filling occurs in first half of diastole) §Decreasing SV §Decreasing myocardial oxygenation potential (coronary arteries fill in diastole)
⬢Type of murmur changes treatment during anesthesia:
§Restrictive conditions require increased filling pressures §Regurgitant conditions are aided by increasing forward flow §Failing myocardium requires contractile assistance
Central Venous Pressure
Monitoring
⬢Zero reference = RA ⬢Measurement in cmH2O ⬢ with a water column ⬢Or in mmHg with ⬢ a pressure transducer ⬢1 mmHg = 1.36 cmH2O ⬢CVP = 0-5 cmH2O in SA ⬢CVP> 12 cmH2O, High ⬢CVP < 0 cm H20, Low
BP cuff should be what % of limb circumference
30-40%
Results of cooling
Increased recovery time: mortality in some studies nDecreased metabolic rate and MAC (5% for each C°) nDecreased oxygen demand of tissues
Capillary Filtration Pressure
•Net Driving Pressure into Capillary •[(Pc-Pi) – (pp- pi)] §Pc = capillary hydrostatic pressure vArteriole approx. 35 mmHg vVenuole approx. 15 mmHg §Pi = interstitial hydrostatic pressure (0) §pp = plasma oncotic pressure (20-28 mmHg) §pi = interstitial oncotic pressure (3mmHg) •Balance » 10 mmHg fluid OUT of capillary • » 10 mmHg fluid IN to venule
Disadvantages of Colloids
•Circulatory overload •Anaphylactic reactions (incidence 0.033%) •Coagulation disorders: §‘Coating of platelets’ §Volume dilution of clotting factors vDextrans (worst) vStarches (hetastarch) vGelatins (not in use)
Assessment of Hydration
⬢Hydration is the presence of fluid in the interstitial space (intravascular and intracellular are far more difficult parameters to assess) §Dehydration for <3 days 80% ECF, 20% ICF §Dehydration for >3 days 60% ECF, 40% ICF ⬢Koch and Graber
Emergency Fluid Resuscitation
⬢Aim: restore tissue perfusion & oxygenation ⬢Shock fluid rates (high end value- constant re-evaluation will most likely limit these numbers): ⬢Think ¼ blood volume to start §Blood volume: vDog 80-90 ml/kg (isotonic) vCat 60-70 ml/kg (isotonic) vColloid (dextran or hetastarch) 20ml/kg §7.5% Hypertonic saline in colloid 4ml/kg over 10min
Acute Blood Loss
⬢Goal: restore circulating blood volume (CBV) ⬢Initially any fluid type will restore volume and maintain cardiac output ⬢Volume of crystalloid must be 3 x volume of colloid for the same ­ CBV ⬢Replace red cells or use whole blood once bleeding is under control, or when loss exceeds 20% of blood volume ⬢What comes out must go in

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