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General Anesthetics (Inhalation)

General anesthetics - inhalation, p. 143-161

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decrease
____ rate of induction: increase solubility of anesthetic in blood (blood/gas partition coefficient), increase cardiac output
nitrous oxide
disadvantages: relatively weak; post-op N&V; hypoxia during recovery; chronic exposure -> vitB12 deficiency
uniquely titratable
general property of inhalation anesthetics: by altering partial pressure in inspired air; moment-to-moment control
pulmonary blood flow
esp. important factor for anesthetics w/ moderate-high blood solubility; INCREASE this, DECREASE rate of induction (b/c more blood for gas to dissolve into)
recovery
affected by: 1) pulmonary blood flow, 2) pulmonary ventilation, 3) solubility of anesthetic in various tissues (+ duration of exposure), 4) solubility of anesthetic in blood (if less soluble, this is faster); metabolism NOT usually a factor
Minimum Alveolar Concentration (MAC)
values INCREASE (potency decreases) with desensitization to CNS depressants (chronic alcoholism), CNS stimulants (e.g. amphetamine, cocaine); INDEPENDENT of gender and weight!
therapeutic index
general property of inhalation anesthetics: this is low, has a low margin of safety (2-4)
sevoflurane
disadvantages: CV and respiratory depression; expensive (b/c new); can be chemically unstable w/ exposed to CO2 adsorbents -> compound that may be nephrotoxic
slow acting
include: enflurane, isoflurane, halothane
fast acting
include: nitrous oxide, desflurane, sevoflurane
isoflurane
advantages: stable cardiac rhythm, quite rapid onset and emergence; minimal metabolism; muscle relaxant; use for MAINTENANCE; most widely used anesthetic in adults!
mole fraction
one way of expressing MAC (the other is partial pressure in alveolar gas); partial pressure as a % of total gas pressure
oil/gas partition coefficient
indicates relative SOLUBILITY of anesthetic in membranes relative to air; directly related to potency
membrane proteins
volatile and IV anesthetics act DIRECTLY on these by binding to specific hydrophobic sites to change their function
dantrolene
drug of choice for prevention and treatment of malignant hyperthermia; skeletal muscle relaxant that blocks Ca release from SR
inspiration
1st step in getting I.A. to brain; affected by concentration in inspired gas, pulmonary ventilation (# respirations- increase that, increase uptake)
systemically depressant
general property of inhalation anesthetics: depress respiratory and CV systems
stage III
stage of anesthesia: "surgical anesthesia"- regular breathing, unconsciousness, decreased eye mvmt; loss of eyelash reflex
malignant hyperthermia
lethal, acute disorder developing during or after general anesthesia; massive increase in metabolic rate caused by uncontrolled Ca2+ efflux from SR -> muscle tetany and ____
cellular effects
1) hyperpolarize neurons, 2) decrease synaptic transmission- modulation of synaptic transmission occurs at lower concentrations, POST-synaptic actions dominate
stage IV
stage of anesthesia: "medullary depression"- respiratory arrest, cardiac depression and arrest, no eye mvmt
glycine receptors
inhalation anesthetics, propofol, barbiturates enhance capacity for these receptors to be activated
presynaptic nerve terminal
actions here: interfere w/ NT release (potentiate inhibitory synapses, inhibit excitatory synapses); alter reuptake
complete anesthetics
general property of inhalation anesthetics: cause loss of consciousness, immobility, amnesia, analgesia, skeletal muscle relaxation at high doses
increased intracranial pressure
toxic effect of inhalation anesthetics, from increased cerebral blood flow; risk in pts. w/ brain tumor or head injury; reduce risk is pt. is hyperventilated before anesthesia
desflurane
advantages: rapid onset, recovery; high potency; minimal metabolism; use for OUTPATIENT anesthesia
stage I
stage of anesthesia: "analgesia"- conscious, but no pain; analgesia, amnesia, euphoria; muscles and airways not relaxed
halothane
disadvantages: slow induction, recovery; high metabolism -> fatal hepatotoxicity, side effects; CV and respiratory depression; arrhythmias
GABA-a receptors
halogenated anesthetics, propofol, barbituates, etomidate increase sensitivity of these receptors, enhance INHIBITORY neurotransmission, depress nervous system activity
intraoperative awareness
patient becomes conscious during procedure, has recall of events; rare occurrence; no pain; risks: genetics, age, cardiac reserve, substance abuse
second gas effect
uptake of a large volume of primary gas (low potency, e.g. N2O) accelerates uptake of a 2nd gas (more potent, smaller amounts)
desflurane
disadvantages: CV and respiratory depression; expensive (b/c new); VERY pungent, irritating (bad for asthma); laryngospasms; tachycardia
blood/gas partition coefficient
indicates relative solubility of anesthetic in blood relative to air; when this is higher, more anesthetic must be dissolved in blood to raise pp in gas phase
isoflurane
disadvantages: CV and respiratory depression; pungent odor; airway irritant (bad for asthma!); coughing, laryngospasms
nitrous oxide
only inhalation anesthetic that does NOT DECREASE respiration, decrease bp, increase PVR
blood flow to brain
increase this, have faster equilibration of CNS w/ alveolar partial pressure
blood/gas partition coefficient
physicochemical property of inhalation anesthetics that predicts RATE OF INDUCTION (how fast it works); ratio of anesthetic concentrations in compartments when pp of anesthetic is equal in them
balanced anesthesia
a combination of drugs is used to provide more satisfactory anesthesia than from just 1 drug - use desirable properties, minimize adverse actions (b/c of lower dose)
lambda
solubility of anesthetic in blood; is primary determinant of rate of onset/offset of anesthesia; affects passage from alveoli to blood in pulmonary capillaries (2nd step in getting I.A. to brain)
lipid solubility
potency of inhalation anesthetics directly correlated to this (Meyert-Overton rule); suggests hydrophobic site of action
stage II
stage of anesthesia: "excitement"- excitement, delirium, combative behavior, irregular breathing
postsynaptic nerve terminal
actions here: interfere w/ NT binding to receptors; interfere w/ ionic conductance
uterine relaxants
include halothane, enflurane, and isoflurane; useful for fetal manipulations; may increase bleeding postpartum or during therapeutic abortion
Minimum Alveolar Concentration (MAC)
the concentration of anesthetic at 1atm which prevents mvmt in 50% of people exposed to noxious surgical stimulus; is an indication of POTENCY (lower this, higher potency); effective anesthetic concentration is 1.2-1.5x this
oil/gas partition coefficient
physicochemical property of inhalation anesthetics that predicts POTENCY; is inverse of MAC value; ratio of anesthetic concentrations in compartments when pp of anesthetic is equal in them
malignant hyperthermia
genetic predisposition (AD inherited disorder- mutations in SR CA channel- ryanodine receptor) and triggering agents (halogenated volatile anesthetics - NOT N2O, depolarizing muscle relaxants) necessary to evoke this
K+ channels
halogenated inhalation anesthetics activate these channels; set resting membrane potential- activation leads to hyperpolarization
sevoflurane
advantages: fast induction, recovery; high potency; less irritating vapor; use for OUTPATIENT anesthesia, induction, esp. in kids
increase
____ rate of induction: increase concentration of anesthetic in inspired-gas mixture, increase alveolar ventilation
nitrous oxide
advantages: no odor; fast induction and emergence; minimal cardiopulmonary depression; good analgesic; use for minor surgery, combination (2nd gas effect)
anesthetics
most dangerous drugs- low therapeutic indices, steep dose-response curves, no pharmacological antagonists to correct high doses
halothane
advantages: pleasant odor, nonirritating vapor; bronchodilator (good for asthma!); use for KIDS; MAINTENANCE of anesthesia; asthma pts.
Minimum Alveolar Concentration (MAC)
values DECREASE (potency increases) with increasing age, pregnancy, hyPOthermia, hyPOtension, adjuvants (e.g. CNS depressants)
NMDA receptors
ketamine, NO, cyclopropane INHIBIT these receptors

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