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Microbial Disease

Mother micro

Terms

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decline
decline of disease
ID50 T. palidum (syphilis)
10,000,000
catalase
enzym made by aerobes and facultative anaerobes to convert H2O2 into harmless O2 + H2); catalase neg. microbes are killed by H2O2
cyto
kills cells (Clostridium diphtheriae, B. pertussis, S. pyogenes
Mutualism
both benefit (E. Coli)
dose (n)
number of microbes
Koch's postulates
association, isolation, causation, reisolation
causation
agent must cause the disease when inoculated into a lab animal
coagulase
walls off bacteria, protecting from phagocytosis (Staph)
stages of disease (contagious during all stages)
incubation, prodromal, illness, decline, convalescence
glycocalyx layer
adhesins of Streptococcus mutans (dental carries)
flaccid paralysis
Botulism
entero
damage GI cells (E. Coli, V. cholerae, S. aureus (food poison))
antigenic variation
change Ag. to evade immune system (flu, trypanosome (African Sleeping Sickness, Chagas), HIV, gonorrhea,
association
agent must be in all with the specific disease
Protozoans with virulence factors
Plasmodium (malaria), trypanosome (African Sleeping Sickness), Giardia (giardiasis dysentery)
Enzymes
coagulase, collagenase, hemolysins, hyaluronidase, kinase, leukocidin, catalase
other adhesins
flagella, hooks, suction disc
leukocidin
destroys WBC
virulence (v)
microbial "weapons" factors
hemolysins
a-partial, b-full (Strep pyogenes), y-no RBC destruction
infection
invasion and multiplication of pathogen
pathogenicity of disease depends on?
dose, virulence, resistance
isolation
agent must be isolated and grown in pure culture
ID50
dose required where 50% will be infected
incubation
after invasion; microbial replication
dental carries
Streptococcus mutans
waxy cell wall
TB
ID50 legionella
1
ID50 salmonella
10
An opportunistic pathogen is generally of ... virulence and requires ... host resistance
low; decreased
sub-clinical
asymptomatic
capsules
adhesin, slimy layer around cell wall decreases phagocytosis (S. pyogenes)
What are the three types of symbiosis?
Commensalism, Mutualism, Parasitism
toxins
exo, endo, cyto, neuro, entero
reisolation
agent must be isolated from all lab animals and be identical to the original microbe
virulence factors
adhesins, toxins, enzymes
exo (heat labile=susceptible to destruction by heat)
strong, made and released during microbial growth, mostly Gm+
resistance
strenth of host's immune system
pneumonia
P. carinii
illness
acute disease
spikes
adhesins, flu, HIV (H + N)
Commensalism
one benefits; the other has no effect
prodromal
symptoms
hyaluronidase
destroys hyaluronic acid of connective tissue (Strep pyogenes)
neuro
damages nerve cells (Clostridium tetani botulinum)
What are Koch's postulates used in?
research
which is worse, food intoxication or food infection?
food intoxication
endo
released as LPS of Gm-cell wall is destroyed, leads to shock, maybe death, heat stable (ex. Salmonella typhi, N. meningitidis)
ID50 is shifted by
increased/decreased virulence or increased host resistance
How many types does symbiosis have?
3
kinase
destroys protective blood clots (evade phagocytosis), streptokinase is used to prevent heart attack and stroke
collagenase
destroys collagen in connective tissue (S. pyogenes, Clostridium perfringens)
Examples of opportunistic infection
PCP in AIDS, candidiasis in AIDS, peritonitis from E.Coli
convalescence
recovery (but can still be a carrier)
What do Koch's postulates prove?
a specific organism causes a specific disease
Example of commensalism
Staphylococcus epidermidis
have waxy M protein in addition to capsules
B. anthracis, Y. pestis, S. pneumoniae
Parasitism
one lives in another and harms it (protozoans, viruses)
LD50
dose required where 50% will die
Pathogenicity formula
D=nR/V
spastic paralysis
Tetanus
pili
adhesin for N. gonorrheae
disease
symptoms due to tissue damage

Deck Info

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