A and P 3
Terms
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- what is the exception with the kidney
- not carbonic acid
- where does angiotensiongen come from
- liver
- body range for ph
- 6.8 to 8
- what do the left areieral receptors do when there is a increase in ECF col arterial pressure greater than
- less vasopressin= not thirsty
- what do the left areieral receptors do when there is a decrease in ECF col arterial pressure greater than 7%
- increase vasopressin= thirsty
- what two source of H come from food
- Hpo3 and H2so4
- above what is alkalsosis
- 7.45 (to 8)
- fxn of renin
- convert angotensiongen to angiotensen1
- ph equation
- = pk + log of (HCo3/Co2)
- how does the hemoglobin buffer system work
- carbonic acid cahnges
- Four chem buffer systems
- H2Co3, HCo3, Protien system, Hemoglobin, Phosphate
- what is the H20 input and out put
- 2600 ml
- sources of H
- carbonic acid Hpo3 H2So4 Facids and Lactic Acid
- fxn of the left arterial receptors
- monitor BP in ECF
- what do the hypothalamous osmoreceptors do wen there is a increase in osmolartiy
- increase vasopressin secretion= thirsty
- effectiveness of respiratory buffere sytem
- 50-75%
- what does the retntion of water and salt turn around and do
- via the juxtaglumular apperatus in the kidneys- decreases renin secretion
- fxn of angiotensin2
- increase sym activity (constriction and increase bp) reabsorb NaCl excret K realse aldosteron and Adh
- pH of urin
- 4.5-6
- aka for ADH
- vasopressin
- long term regulation of fluid balance
- kidney and thirst
- fxn of ECF osmolartiy
- prevent shirking/sweeling via water balance
- what happens to aldosterone when there is a decrease in BP due to low sodium
- increase in aldosterone which increases NA resabsorption which increase bp
- dieases associated with hypotonicity
- renal failure because more vasospressing to increase retention and excess water
- Consequences of a flux in ph
- change exciability of nerve and msucles, change enzyme activity, change k levels in body
- fxn of ECF volume
- maintain bp via salt balance
- where does the H2Co3 and HCo2 buffere system work
- ECF for noncarbonic acid (abundant)
- how does the respiratory buffer system work
- by removal of Co2
- where does ACE come from
- lungs and kidney
- where is vasporessin stored i
- in the pituitary gladn
- what is resposible foor the secretion of ammonia
- glutamine
- what do the hypothalamous osmoreceptors do wen there is a decrease in osmolarity
- decrease vasopressin = not thirsty
- 4 compensation mechanism for ph
- chem buffer, respiration and kidneys
- fxn of ace
- go from 1 to 2
- where do the phosphate buffere system work
- urniary and the icf
- short term regulation of fluid blanace
- baroreceptors/fliud shifts
- relationship btw bp and hydration
- low bp with lower hydration
- where does renin come from
- kidney
- tonicity related to diabetes insipidu
- decrease vasopressin therefore no kindy absorbtion
- relationship btw vasopressin and thirst
- vasopressing makes you thirsty
- equation for ht phsophate buffer system
- na2Hpo4 + H <--> NaPH2Po4 + NA
- where does ADH come from
- pitutiary gland
- when there is a decrease is salt what happens to bp
- decreases
- where does the protien buffere system work
- both ICF and ECF
- when is ther no kidney compensation
- with metabolic acidosis Renal failur urimica
- what is alway sknow as the thirst center
- hypothalamus which makes vasopressin
- where does aldosteron come from
- adrenal gland
- ph of NH4 in the urine?
- 4.5 or greater
- what does the kidney excrete to buffer
- H, Hco3, Amonia
- below what is acidosis
- 7.35 (to 6.8)
- when blood pressure decreases do to low salt wat happens to glomeluar filtration
- less plasma filters Na therefore less na is excreted which raises blood pressure
- what makes vasopressin
- hypothalamus
- what are three things angiotension causes the reslease of to retain salt and water
- nacl reabsorption, release of aldoseron and ADH