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A and P 3

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what is the exception with the kidney
not carbonic acid
where does angiotensiongen come from
liver
body range for ph
6.8 to 8
what do the left areieral receptors do when there is a increase in ECF col arterial pressure greater than
less vasopressin= not thirsty
what do the left areieral receptors do when there is a decrease in ECF col arterial pressure greater than 7%
increase vasopressin= thirsty
what two source of H come from food
Hpo3 and H2so4
above what is alkalsosis
7.45 (to 8)
fxn of renin
convert angotensiongen to angiotensen1
ph equation
= pk + log of (HCo3/Co2)
how does the hemoglobin buffer system work
carbonic acid cahnges
Four chem buffer systems
H2Co3, HCo3, Protien system, Hemoglobin, Phosphate
what is the H20 input and out put
2600 ml
sources of H
carbonic acid Hpo3 H2So4 Facids and Lactic Acid
fxn of the left arterial receptors
monitor BP in ECF
what do the hypothalamous osmoreceptors do wen there is a increase in osmolartiy
increase vasopressin secretion= thirsty
effectiveness of respiratory buffere sytem
50-75%
what does the retntion of water and salt turn around and do
via the juxtaglumular apperatus in the kidneys- decreases renin secretion
fxn of angiotensin2
increase sym activity (constriction and increase bp) reabsorb NaCl excret K realse aldosteron and Adh
pH of urin
4.5-6
aka for ADH
vasopressin
long term regulation of fluid balance
kidney and thirst
fxn of ECF osmolartiy
prevent shirking/sweeling via water balance
what happens to aldosterone when there is a decrease in BP due to low sodium
increase in aldosterone which increases NA resabsorption which increase bp
dieases associated with hypotonicity
renal failure because more vasospressing to increase retention and excess water
Consequences of a flux in ph
change exciability of nerve and msucles, change enzyme activity, change k levels in body
fxn of ECF volume
maintain bp via salt balance
where does the H2Co3 and HCo2 buffere system work
ECF for noncarbonic acid (abundant)
how does the respiratory buffer system work
by removal of Co2
where does ACE come from
lungs and kidney
where is vasporessin stored i
in the pituitary gladn
what is resposible foor the secretion of ammonia
glutamine
what do the hypothalamous osmoreceptors do wen there is a decrease in osmolarity
decrease vasopressin = not thirsty
4 compensation mechanism for ph
chem buffer, respiration and kidneys
fxn of ace
go from 1 to 2
where do the phosphate buffere system work
urniary and the icf
short term regulation of fluid blanace
baroreceptors/fliud shifts
relationship btw bp and hydration
low bp with lower hydration
where does renin come from
kidney
tonicity related to diabetes insipidu
decrease vasopressin therefore no kindy absorbtion
relationship btw vasopressin and thirst
vasopressing makes you thirsty
equation for ht phsophate buffer system
na2Hpo4 + H <--> NaPH2Po4 + NA
where does ADH come from
pitutiary gland
when there is a decrease is salt what happens to bp
decreases
where does the protien buffere system work
both ICF and ECF
when is ther no kidney compensation
with metabolic acidosis Renal failur urimica
what is alway sknow as the thirst center
hypothalamus which makes vasopressin
where does aldosteron come from
adrenal gland
ph of NH4 in the urine?
4.5 or greater
what does the kidney excrete to buffer
H, Hco3, Amonia
below what is acidosis
7.35 (to 6.8)
when blood pressure decreases do to low salt wat happens to glomeluar filtration
less plasma filters Na therefore less na is excreted which raises blood pressure
what makes vasopressin
hypothalamus
what are three things angiotension causes the reslease of to retain salt and water
nacl reabsorption, release of aldoseron and ADH

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