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CNS drugs

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partial opioid agonist at mu receptors, agonist at kappa receptors
butorphanol
weak opioid agonist that also inhibits 5-HT and NE uptake
tramadol "tram it all" in
On opioids, tolerance develops to everything but...
constipation and miosis
dec aqueous humor synthesis via vasoconstriction
alpha agonists -EPI - do NOT use in closed-angle glaucoma -brimonidine
dec aqueous humor secretion
B blockers - timolol, betaxolol, carteolol acetazolamide
inc outflow of aqueous humor
cholinomimetics (contract ciliary muscle and open trabecular meshwork) -pilocarpine (emergencies) -carbachol -physostigmine -echothiophate PG - latanoprost (PGF2a) The circular ciliary muscle fibers affect zonular fibers in the eye (fibers that suspend the lens in position during accommodation), enabling changes in lens shape for light focusing. When the ciliary muscle contracts, it pulls itself forward and moves the frontal region toward the axis of the eye. This releases the tension on the lens caused by the zonular fibers (fibers that hold or flatten the lens). This release of tension of the zonular fibers causes the lens to become more spherical, adapting to short range focus. The other way around, relaxation of the ciliary muscle causes the zonular fibers to become taut, flattening the lens, increasing long range focus. Contraction and relaxation of the longitudinal fibers, which insert into the trabecular meshwork in the anterior chamber of the eye, cause an increase and decrease in the meshwork pore size, respectively, facilitating and impeding aqueous humour flow into the canal of Schlemm.
darkens color of iris
latanoprost
anticonvulsants with liver toxicity
carbamazepine valproic acid
first line for tonic-clonic seizures
phenytoin carbamazepine valproic acid
contraindicated in pregnancy (teratogenic)
phenytoin carbamazepine valproic acid
first line in pregnant women, kids
phenoBarbital
used for trigeminal neuralgia
carbamazepine
used for neuropathic pain
gabapentin
causes Stevens-Johnson syndrome
ethosuximide lamotrigine
used for absence seizures
ethosuximide (first line) valproic acid
Prophylaxis for status epilepticus
Phenytoin
acute status epilepticus
BDZ
induce cyt P450
phenytoin carbamazepine valproic acid
causes wt gain
valproic acid
causes wt loss
topiramate
kidney stones
topiramate
agranulocytosis, aplastic anemia
carbamazepine
megaloblastic anemia hirsutism SLE-like syndrome gingival hyperplasia
phenytoin
mental dulling
topiramate
inc Na inactivation
phenytoin - use dependent blockade, also inhibits glutamate release carbamazepine
inc GABA-A action
phenobarbital - inc duration of Cl channel opening (barbiDURATE) BDZ - inc frequency of Cl channel opening (FREnzodiazepine)
block thalamic T-type Ca channels
ethosuximide
BDZs for status epilepticus
lorazepam diazepam Lora Diaz had epilepsy.
short-acting BDZs highest addictive potential
triazolam oxazepam midazolam TOM thumb
GABA antag to treat BDZ overdose
flumazenil
The more lipid-soluble a drug is, the ____ the MAC and the ____ the potency.
lower higher
low blood solubility
rapid induction and recovery
N2O has _____ blood solubility and _____ lipid solubility.
low,low fast induction and low potency
Halothane has ____ blood solubility and _____ lipid solubility.
high, high high potency, slow induction
inhaled anesthetics
halothane "fluranes" N2O
halothane
hepatotoxicity
enflurane
proconvulsant
methoxyflurane
nephrotoxicity
inhaled anesthetics SE
malignant hyperthermia inc cerebral BF myocardial and respiratory depression nausea/emesis
IV anesthetics
barbiturates - thiopental BDZ - midazolam ketamine opiates - morphine, fentanyl propofol "B B King on OPIATES PROPROses FOOLishly"
rapid induction, short procedures
thiopental - DEC cerebral BF propofol
most common drug for endoscopy
midazolam (used with inhaled anesthetics and narcotics) may cause sevrer postop resp depression, dec BP, amnesia
block NMDA Rs
ketamine PCP analog cardiac stimulant disorientation, hallucination, bad dreams INC cerebral BF
local anesthetics
esters - procaine, etc. amides - lidocaine, bupivacaine, etc. can cause CNS excitation, severe CV toxicity (bupivacaine), HTN, hypoTN, arrhythmias (cocaine) except for cocaine, given with vasoC (EPI) to dec bleeding, dec systemic concentration in infected (acidic) tissue, alkaline anesthetics are charged and cannot penetrate membranes effectively. more anesthetic is needed in these cases. preferentially bind activated Na channels
order of nerve blockade
small faster than large myelinated fasted then unmyelinated size factor predominates: 1. small myelinated 2. small unmyelinated 3. large myelinated 4. large unmyelinated
order of sensory loss
pain temp touch pressure PTTP
depolarizing NM blocker
succinylcholine -can cause hyperCa, hyperK -phase I: prolonged depol, no antidote, cholinesterase inhibitors potentiate blockade -phase 2: repolarized but blocked. use cholinesterase inhibitors
competitive NM blockers
tubocurarine, atracurium, pancuronium, etc.
treat malignant hyperthermia, malignant neuroleptic syndrome
dantrolene - prevent Ca release from SR
drugs for Parkinson's
Bromocriptine, pramipexole, ropinirole - DA agonist Amantadine - inc DA release, can cause ataxia L-dopa/carbidopa - can cause arrhythmias (peripheral conversion), dyskinesia, akinesia Selegiline (MAO-B inhibitor), entacapone, tolcapone (COMT inhibitors) Antimuscarinic - benztropine - helps with tremor and rigidity but not bradykinesia Al Capone hates commies decrease your tremor before you drive your BENZ
memantine
NMDA antag used to treat AD SE: confusion, dizziness, hallucinations
donepezil
anticholinesterase used to treat AD SE: dizziness, nausea, insomnia
sumatriptan
5-HT1B/1D agonist causes vasoC, inhibition of trigeminal activation and vasoactive peptide release t1/2 less than 2 hrs treat: migraine, cluster headaches SE: coronary vasospasm (don't use in ppl with CAD, Prinzmetal's angina), tingling
INC cerebral blood flow
inhaled anesthetics ketamine
DEC cerebral blood flow
thiopental
CV toxicity
bupivacaine

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