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Chapter 16 Final 2

Terms

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Involves histamine as an important mediator
Type I and III hypersensitivity
Can be induced by poison oak in sensitive individuals
Type IV hypersensitivity
Can lead to asthma
Type I hypersensitivity
Occurs as result of mismatched blood transfusions
Type II hypersensitivity
Systemic form of reaction is treated with epinephrine
Type I hypersensitivity
Can be induced by pollens and certain foods in sensitive individuals
Type I hypersensitivity
May involve cell destruction by antibody dependent cell mediated cytoxicity
Type II hypersensitivity
One form of clinical manifestation is prevented by Rhogam
Type II hypersensitivity
Localized form characterized by wheal and flare reactions
Type I hypersensitivity
IgE mediated degranulation of mast cells
Type I hypersensitivity
Lysis of antibody coated blood cells by complement
Type II hypersensitivity
Tissue destruction in response to poison oak
Type IV hypersensitivity
C3a and C5a mediated mast cell degranulation
Type III hypersensitivity and some type II
Chemotaxis of neutrophils
Type III hypersensitivity
Chemotaxis of eosinophils
Type I hypersensitivity
Activation of macrophages by IFN-gamma
Type IV hypersensitivity
Deposition of antigen-antibody complexes on basement membranes of capillaries
Type I hypersensitivity

Deck Info

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